goodlife4casey wrote:
Take the med and kill the euphoria of the drinking; but stop drinking and other things that are enjoyable are enhanced because you’re off the meds.
From what I am seeing, the Nal is not stopping any sensible euphoria. By sensible, I mean a feeling that we can experience. I think it is effecting something a little more subliminal. At least for me.
Last night I drank enough to get drunk. Other than doing it over a much longer period (about 4 hours), I think I experienced everything I ever had EXCEPT, the accelerator never kicks in. I used to drink faster and faster until I got drunk but now I actually seem to slow down. I had six drinks of gin last night and went to bed. Before I would not know how many I drank but would have woke up on the couch.
I don't think we sense endorphins directly. We taught ourselves to release them for a particular behavior and when they are released, the are picked up by receptors and the cycle is complete. But when the cycle is not complete, we translate that with as need to drink. If we think that drinking is probable, we experience excitment. When we anticipate that we won't be able to drink we trigger a craving to please those receptors. We only translate that as a need to drink. That is the reason that gambling addicts experience the identical craving even though no external substance it apparent.
By taking the Naltrexone, we allow the process to take place up until its final phase, endorphin binding with opioid receptor. But since we had learned that drinking would stop the craving, we have been tricked. But we are only able to trick ourselves so long and then the brain says, "I ain't gonna dump these precious endorphins if they have no where to go". This is atrophy. And the learning is extinguished.
Pavloc has rang the bell, the dog has salivated but no treat was giving. At some point the dog quits drooling and eventually attacks the owner. Don't try this at home with a pit bull.